Utilize este identificador para referenciar este registo:
|Título:||The genetics of non-host disease resistance in wheat to barley yellow rust|
|Citação:||Rodrigues, Paula; Garrood, J.M.; Shen, Q.-H.; Smith, P.H.; Boyd, L.A. (2004). The genetics of non-host disease resistance in wheat to barley yellow rust. TAG Theoretical and Applied Genetics. ISSN 0040-5752. 109:2, p. 425-432|
|Resumo:||Non-host resistance is investigated as a potential source of durable resistance. However, the genetics of non-host resistance between closely related plant species and their corresponding pathogens would indicate that in these interactions, non-host resistance primarily involves major genes that operate on a gene-for-gene principal similar to that seen in host resistance. Wheat is a non-host of the barley-attacking form of the fungus responsible for yellow rust, i.e. Puccinia striiformis f. sp. hordei. While P. striiformis f. sp. hordei is generally unable to infect wheat, a partial susceptibility was exhibited by the wheat variety Chinese 166. Consequently, in the cross Lemhi × Chinese 166 two major QTLs for resistance to P. striiformis f. sp. hordei were identified: one on chromosome 1D and a second on 2B. These two QTLs accounted for 43.5% and 33.2% of the phenotypic variance for resistance to barley yellow rust, respectively. In addition, two QTLs of smaller effect were also identified: one on chromosome 5A, contributing 5.1% of the variance and a second on chromosome 6A, contributing 10.9% to the phenotype. The QTL on 6A was derived from the susceptible variety, Chinese 166. In all cases the resistance towards P. striiformis f. sp. hordei was associated with a visual chlorosis/necrosis response typical of race specific host resistance.|
|Aparece nas colecções:||CIMO - Artigos em Revistas Indexados à WoS/Scopus|
Ficheiros deste registo:
|TAG109.pdf||162,02 kB||Adobe PDF||Ver/Abrir Acesso Restrito. Solicitar cópia ao autor!|
Todos os registos no repositório estão protegidos por leis de copyright, com todos os direitos reservados.